Terminology. The term cortical laminar necrosis is often used to describe areas of cortical T hyperintensity or cortical dystrophic calcification in the weeks, months, or years following a race. Surgical decision-making in adult spondylolisthesis is complex and can be divided into degenerative, low-grade lytic spondylolisthesis. listhesis and high-grade listhesis. 4.1. Degenerative spondylolisthesis. Degenerative spondylolisthesis is the most common cause of spondylolisthesis in adults. GII L4-L, 18 scoliosis: Stable listhesis, progressed scoliosis, 4: L4-S: L3-pelvis PSF: 243: GI L5-S: Stable listhesis, worsening of foraminal stenosis: 5: L4-L5, L5-S : L5-S: 625: GI L5-S: Listthesis of progression to GII: 6: L3-L4, L5-S: L4 - 728: No, Physiotherapy. During the acute phase of rehabilitation of patients with spondylolysis, emphasis is placed on pain reduction. Teaching posture and biomechanics along with ADL activities of daily living can help protect injured parts, thereby reducing symptoms and preventing further injury. A rest period for an average - can. Lumbosacral spondylolisthesis is the forward translation of the fifth lumbar vertebra L5 on the first sacral vertebra S1. Bilateral L-shaped spondylolysis or repetitive stress injury is the main etiology of lumbosacral spondylolisthesis. The degree of slippage often correlates with the degree of symptoms. The most common symptom of anterolisthesis is lower back pain. The pain may radiate to the buttocks and back of the thighs. Other symptoms may include: back stiffness. muscle spasms. tight hamstrings. numbness, tingling, or weakness in one or both legs. difficulty walking. What does ls anterolisthesis mean for bilateral pars defects. Spondylolisthesis: Isthmic spondylolisthesis is another term for what you are describing. Basically, one of the vertebrae is offset from the other. What caused this condition in your case is a defect in the pars interarticularis, which is usually the case. The transition from a thoracolumbar facet with a sagittal orientation to an L5-S joint with a coronal orientation explains the predisposition to dislocation and listhesis. 5, 25- robust lower lumbar facets and stronger ligamentous attachments of the lower lumbar spine, disruption of these structures and the presence of a sacral slope leads to a,